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Erratum

In the article by P. R. Devchand et al. that starts on page 652 of Volume 17, Number 6, of The FASEB Journal, two figures were transposed and the gradation between gray and black were not apparent. The figures and captions are reproduced below:

View larger version (21K): [in this window] [in a new window]   Figure 6. Myeloid expression of hALX changes the exudate levels of LXA4 and LTB4. Upper left panel: LC/MS/MS of endogenous LXA4 in peritoneal exudates from hALX transgenic mice (black bars, n=2) and their nontransgenic littermates (gray bars, n=2). Exudates formed after peritoneal injection of zymosan A for 4 h, as in Fig. 4. The proinflammatory eicosanoid LTB4 (lower panel left) in nontransgenic mice (gray bars) and in transgenic hALX mice (line A, black bars). Upper right: MS/MS spectra of LXA4 (M-H, m/z 351); lower right: LTB4 (M-H, m/z 335). Diagnostic ions are denoted in the inset structures of LXA4 and LTB4 (using gray arrows) with major fragments indicated in their respective spectrum. Also, LC retention times and characteristic UV chromophores were used for identification of each eicosanoid (see text and Materials and Methods for further detail).

View larger version (19K): [in this window] [in a new window]   Figure 7. ATLa inhibition of zymosan-induced murine peritonitis in ALX transgenic and nontransgenic littermates. ATLa (indicated amounts in 120 µl saline) was injected by intravenous tail vein injection, followed by zymosan A (1 mg in 1 ml saline) installation into the peritoneum. Mice were sacrificed (2 h), peritoneal lavages collected and cells enumerated. Inset: % inhibition with ATLa (10 ng) for ALX vs. nontransgenic littermates. Results represent SE for 3 or 4 different sets of transgenic (line B) and nontransgenic littermates. *Statistically significant difference (p < 0.05); #Not statistically significant (TG vs. non-TG) at 1 µg i.v.

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