FASEB J.
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Figure 2


Figure 2. Schematic representation demonstrating that the Merg1a channel is up-regulated in response to skm disuse and that channel function yields an increase in the ubiquitin proteasome proteolysis that participates in the atrophic process. Two obvious areas of needed research are highlighted by questions marks: 1) by what process does disuse signal the up-regulation of Merg1a protein; and 2) by what mechanism does Merg1a function signal UPP activity.





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